tio2 water treatment

In vitro, in the hemocytes of the marine mussel Mytilus hemocytes, suspension of TiO2 NPs (Degussa P25, 10 μg/ml) stimulated immune and inflammatory responses, such as lysozyme release, oxidative burst and nitric oxide production. Vevers and Jha demonstrated the intrinsic genotoxic and cytotoxic potential of TiO2 NPs on a fish-cell line derived from rainbow-trout gonadal tissue (RTG-2 cells) after 24 h of exposure to 50 μg/ml. Reeves et al. demonstrated a significant increase in the level of oxidative DNA damage in goldfish cells, and suggested that damage could not repaired by DNA repair mechanisms. Another suggestion from the mentioned study was that hydroxyl radicals are generated also in the absence of UV light. It has been shown that fish cells are generally more susceptible to toxic/oxidative injury than mammalian cells.

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The first commercial production of TiO2 began in the early 20th century, using the sulfate process. This method involved reacting ilmenite ore with sulfuric acid to produce titanium sulfate, which was then calcined to obtain titanium dioxide. However, this process had several drawbacks, including high energy consumption, generation of large amounts of waste, and release of harmful gases such as sulfur dioxide. As a result, many factories transitioned to the chloride process, which offers higher purity TiO2 and reduced environmental impact.

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